how much is fireworks disney dessert

ore!As the fireworks begin, celebrate with a sparkling cider toast and create a special memory with family and friends. Throughout the evening, you can now enjoy hot drinking chocolate and signature lemonades as well as coffee and tea. Wishes Fireworks Dessert Party is $49 for adults, $29 for children, tax inclusive. Reservations can be made beginning on March 20 by visiting Disney World.com/dine, or calling 407-WDW-DINE.

Thu, March 19, 2015Reservations Open March 20 for the Wishes Fireworks Dessert Party at Magic Kingdom Parkby Rachel Bshero, Food & Beverage Marketing Communications Coordinator Imagine enjoying an exclusive, unforgettable view of the fireworks over Cinderella Castle while you indulge in a decadent dessert experience. The newly enhanced Wishes Fireworks Dessert Party at Magic Kingdom Park allows you to do just that. Beginning July 5, 2015, Tomorrowland Terrace Dessert Party at Magic Kingdom Park will be enhanced and transformed into the Wishes Fireworks Dessert Party. Relax in a reserved seating area with family and friends over delicious desserts and beverages. Gallery Reservations Open March 20 for the Wishes Fireworks Dessert Party at Magic Kingdom Parkview all Delight in a selection of themed desserts prepared daily by our pastry chef including chocolate-dipped strawberries, chocolate mousse, and ice cream. Delectable new additions to the menu include a variety of seasonal fruit and cheese, Disney character-themed cupcakes, and m

Remember, you can also make your next visit to Epcot extra sweet by making a reservation to enjoy the Illumi Nations Sparkling Dessert Party during the Epcot nighttime fireworks spectacular.


how ford fusion pre collision assist with pedestrian detection works

s. Pre-Collision Assist with Pedestrian Detection*If the system’s sensors detect a potential collision with a car or pedestrian in front of you, a warning flashes on the windshield and an audible alert sounds. Also, the brakes will precharge and increase brake-assist sensitivity to provide full responsiveness when you brake.

Feel confident at the wheel. Feel confident at the wheel. *While Pre-Collision Assist with Pedestrian Detection may be especially helpful in unexpected situations, it does not replace the driver and has limitations, including nighttime, low and harsh lighting conditions, vehicles moving in a different direction and certain weather condition

If the driver does not take corrective action and collision is imminent, brakes can automatically apply. Available on SE, Titanium and Sport and standard on Platinum. *Driver-assist features are supplemental and do not replace the driver's attention, judgment and the need to control the vehicle. While Pre-collision Assist with Pedestrian Detection may be especially helpful in unexpected situations, it does not replace the driver and has limitations, including nighttime, low and harsh lighting conditions, vehicles moving in a different direction and certain weather conditions. Enhanced active park assist with parallel and reverse perpendicular parking New for 2017 and a class exclusive**Class is Midsize Hybrid Sedans, Midsize Sedans and Midsize Plug-In Hybrid Sedans based on Ford segmentation. PERPENDICULAR PARKING ASSISTAvailable Enhanced Active Park Assist 10 allows Fusion to help you with perpendicular parking too. Utilizing 12 ultrasonic sensors, Fusion will scan for suitable parking spaces and can assist in backing the vehicle into the spot. All you have to do is shift, accelerate and brake, and Fusion can take care of the steering. PARK OUT ASSISTFusion is ready to help you steer your vehicle out of a tight parallel parking spot with park out assist. When you’re wedged into a parallel parking space, simply press the button and the system will assist you with instructions on shifting as well as moving forward and back while it steers you out. SIDE PARKING SENSORStay aware of your surroundings with side parking sensor. Fusion warns you when your vehicle becomes too close to objects. BLIS® (Blind Spot Information System)Radar in the rear quarter panels detects whether a vehicle is in your blind spot and alerts you with an indicator light in the appropriate sideview mirror. 10Cross-Traffic Alert Available cross-traffic alert* uses radar to watch for traffic behind you when you’re slowly backing out of a parking spot or your driveway. If cross-traffic alert sensors detect a vehicle approaching from up to 15 yards away, you will be alerted by a visual display in the corresponding mirror and a signal in the message center. You will receive an audible alert as well. Cross-traffic alert comes with BLIS (Blind Spot Information System). Inflatable Rear Safety Belts The Fusion is the first vehicle in its class* to offer inflatable rear safety belts. These available rear outboard safety belts help distribute crash-force energy across more of the torso area than traditional belts, which can help reduce the risk of injury, while providing support to the head and neck. The inflatable rear safety belts are designed to hold the passenger in the appropriate seating position, helping to reduce risk of injury. These safety belts are compatible with many infant and child-safety booster seats. *Class is Midsize Hybrid Sedans, Midsize Sedans and Midsize Plug-In Hybrid Sedans based on Ford segmentation. Adaptive Cruise Control with Stop-and-Go When Fusion is in cruise mode, a radar sensor detects slowing traffic ahead and can automatically brake and accelerate to keep you moving with the pace of traffic 10 . With the addition of stop-and-go, the feature can bring your vehicle to a complete stop in stopped traffic and restart it as traffic begins to move Standard Rear View Camera*Put the gear selector in Reverse and the rear view camera automatically transmits the image of what’s behind your vehicle to your touchscreen when you’re slowly backing up. The rear view camera is standard on all models. *2017 model year shown, with available SYNC® 3 with 8-inch screen, 4.2-inch is standard. Driver-assist features are supplemental and do not replace the driver’s attention, judgment and need to control the vehicle. Lane-Keeping System The Lane-Keeping System, which includes both the lane-keeping alert and the lane-keeping aid can help. A camera mounted behind the windshield scans road lane markings to determine vehicle position and detect a lane departure. The system can operate during the day or night. Lane-keeping alert warns you through a series of steering wheel vibrations, which mimic a rumble strip, should the system detect an unintentional lane departure. Lane-keeping aid actively applies steering torque, which alerts you to direct your vehicle back into the target lane should the system detect an unintended lane departure. 10Lane-Keeping System does not control steering. Previous Technology Next Design


what station is near river street in hoboken, nj

Turn left at 14th Street, traveling 0.2 mile to its end at Hudson Street. Turn right on Hudson Street and continue 1.2 miles to Hudson Place. The PATH Hoboken station is to your left on Hudson Place at River Road. From NJ Turnpike I 95 South follow signs for Lincoln Tunnel. Exit at Interchange 17 Lincoln Tunnel/ Secaucus. Follow signs for I 495 Lincoln Tunnel and then follow directions in the preceding paragraph.

Hoboken Station

Hoboken Station Click service map to enlarge Click here to launch transit on Google Maps for step-by-step transit directions. Location1 Hudson Place, Hoboken, NJ 07030. Served by Newark-World Trade Center and Hoboken-World Trade Center lines. From NJ 3 East follow signs for I 495 Lincoln Tunnel. After passing Kennedy Boulevard Exit, stay in right lane and turn off Hoboken /Weehawken Exit. Stay to right at first traffic light and continue down the hill. Turn right at the bottom of the hill, following the signs for Hoboken. Continue on this road for 1 mile until you can go no further (at 14th Street).

From NJ Turnpike I 95 North and I 78 in the Newark Liberty International Airport area, follow signs for I 78 Holland Tunnel. Travel on I 78 to Exit 14C Holland Tunnel. After paying toll, follow I 78 toward traffic light (Jersey Avenue), turn left and continue 0.4 mile. Just after passing under the underpass, turn right onto Newark Avenue and continue 0.7 mile ahead alongside the railroad to the end of the road where you must turn left at Hudson Street. Then turn right at the next corner onto Hudson Place. The PATH station is one block ahead on your right on Hudson Place at River Road. From US 1 and 9 follow signs for Holland Tunnel. Stay in left lane approaching Holland Tunnel tolls. At first traffic light (Jersey Avenue), turn left and follow instructions in preceding paragraph. Connections Served by Hoboken - 33rd Street and Hoboken-World Trade Center lines Connect with NJ Transit trains to western and northern New Jersey; NJ Transit Hudson-Bergen Light Rail; Metro North trains to Rockland and Orange Counties or ferry to World Financial Center, Pier 11/Wall Street or West 38th Street with NY Waterway bus connections. Accessibility This station is ADA Accessible. To learn more, click here. Nearby Attractions Minutes from Stevens Institute of Technology, Hoboken Museum, Hoboken City Hall and numerous boutiques and cafes. Adjacent to privately owned commuter parking lots. Parking Facilities For Parking Facilities near this station, click here. Amenities Mobile Qubes – Mobile device charging kiosks, Mobile Qubes, have been installed at six PATH stations: Newark, Journal Square, Hoboken, 33 Street, Exchange Place, and Newport. Passengers can rent from and return small battery packs to the kiosks after powering up their smartphones and tablets. Devices rented from a PATH kiosk can be returned to any Mobile Qubes kiosk, including those at the Port Authority Bus Terminal, and at other locations at New York Penn Station. For more information, please visit www.mobilequbes.com/support/. Mobile Qube customer service is available seven days a week, 24 hours a day. Call toll free 800 266-0425.


calculate days past ovulation

ed. DPO (Days Past Ovulation) on Chart The DPO row is shown on the BBT chart if you indicated you ovulated during the cycle you are editing/viewing. Days Past Ovulation (Days 1 to N), also referred to as your luteal phase, is the number of days following ovulation up to the day before your next cycle starts.

Coverline on BBT Chart

Coverline on BBT Chart A horizontal coverline can be automatically drawn on your BBT chart based on the cycle day you indicate you ovulated. The coverline is drawn as follows: The BBT temperatures for the 6 days prior to ovulation are read for this cycle. Five of the six must have temps recorded; One discard (or non-recorded temp) is allowed. The coverline is drawn 0.1 (Fahrenheit) or 0.05 (Celsius) of a degree above the highest temp. The coverline is useful to easily see your pre-ovulatory and post-ovulatory temperatures. In a typical biphasic (2 phased) BBT chart, temps are lower prior to ovulation, and higher after ovulation. Ovulation Shown on Chart A vertical line is drawn on your BBT chart on the day you indicated you ovulat

The day after ovulation is DPO 1, the second day after ovulation is DPO 2, etc. In addition to showing the DPO count, this section of the chart (to the right of the ovulation line) is color coded, so you can easily view your luteal phase. Ovulation Day To indicate you ovulated, use the I Ovulated Today checkbox on the Fertility Edit screen for the date on which you detected ovulation. If you accidentally mark more than one ovulation day in a cycle, the earliest date is used for displaying DPO and the coverline, and all others are ignored. Note: Even if you record your ovulation date, if there are not at least 5 days with temperatures prior to the ovulation day in your cycle, the coverline is not drawn. The vertical ovulation line and DPO will be shown on your chart, even if the coverline can not be drawn. This is particularly useful if you are using OPKs and fertility monitors to determine ovulation, since you can see your luteal phase and its length right on your chart.


how big is magic kingdom in acres

Epcot uses 300 acres, Magic Kingdom about 100, 154 for Disney’s Hollywood Studios, and 403 acres in Animal Kingdom, not counting the parking lots. Number of parks: No other theme park resort has four major parks, and two water parks and no one else in the world competes with Walt Disney World on this point.

18 Numbers That Tell Us How Big Walt Disney World Is Posted by: Cas

sie By Cassie Number of guests: Walt Disney World is the leading theme park in the world, based on park attendance reaching 18.6 million for 2013. Magic Kingdom remains the number 1 park in the world based on guest numbers at 18.6 million, Epcot had 11.2 million followed by Animal Kingdom at 10.2 million, and Hollywood Studios at 10.1 million.

The total for all 11 Disney Parks across the globe was 132.5 million in 2013, making it more than double that of its closest competitor, helping to bring them 2.2 million in profits corporate wide.

Number of acres: 27,258. Walt Disney World covers 47 square miles of Central Florida. (This is about the size of San Francisco or two Manhattan’s.) Walt felt like Disneyland was hemmed in by having no buffer land between his beloved park and the rest of the city around it.

He wanted buffer space and plenty of land to build on when purchasing land for his “Florida Project”. The land was purchased at a price of $5,018, 770. It is the largest theme park resort in the world. Only 5900 acres have been developed thus far. It took 52 months to build the 1971 opening day version of Walt Disney World (18 months of that was just for moving dirt to prepare the site). 850 acres are covered by the lakes and waterways systems.

Number of attractions: Over 139 attractions are listed resort wide on the official Walt Disney World website. Number of shows or forms of entertainment: There are 122 venues resort wide. Number of hotels: There are 30 resorts located on Walt Disney World property offering guests anything from a tent campsite to a luxurious Villa. Each resort is Disney-themed, some from famous Disney movies, sports-themed, futuristic, Caribbean, northeast coastline, you name it and there is probably a Disney resort you will enjoy being immersed in. Number of golf courses: There are 4 golf courses, golf lessons, and three miniature golf courses on Walt Disney World property. This is the most on any Disney property worldwide. There are 77 opportunities listed under Sports & Recreation at the WDW resort. Number of dining locations: There are over 310 resort-wide, counting food carts!Number of transportation options: Approximately 750 water crafts puts WDW in 5 th place in number of watercraft employed in a single location across the world; 270 buses, that travel over 15 million miles a year; each bus driver goes through several weeks of training before driving alone. They must have all the miles of roads and routes memorized because they do not just drive one route, they drive them all, as they are needed. Many parts of the routes are not marked or lighted at night so it is easy to get lost if you have not mastered them. Around 23 trams operate in the Disney World parking lots. Number of Cast Members: Walt Disney World is the largest employer in any single location, with over 65,000 Cast Members who fill the 3000 roles it takes to present the magic at the Walt Disney World resort. For a no obligation, FREE Quote with new bookings contact our sponsor Magical Vacation Planner by calling: 1 (407)442-0289 Or for a free no obligation quote with new bookings you can fill out the form by Clicking HERE!Number of park operating hours this is a number I would like to know, but have not found. Taking into account days the park hours are extended due to increased crowd level, Extra Magic Hours, Parties, 24-hour events, I believe Walt Disney World has to be at the top of the list. My 2 nd favorite part as a Guest is when I hear that rare public announcement throughout the park stating the hours have just been extended for the day to give us even more time to enjoy the attractions. My favorite experience is when the park is open 24 hours for a special event. Number of free things: FP+ is included with admission to the Walt Disney World parks at no additional charge, unlike some other Orlando theme parks; Extra Magic Hours and complimentary Magic Bands for WDW resort guests; games within the Parks- Pirates Adventure-Treasures of the Seven Seas and Sorcerers Apprentice at Magic Kingdom; Agent P’s World Showcase Adventure game at Epcot; Wilderness Explorers Club at Animal Kingdom; don’t forget the all-important free ice water at any counter service location; samples at Goofy’s Candy Company and Main Street Confectionary; Club Cool with free samples of soft drinks from around the world; complimentary drawing from Animation Academy where you learn to draw a Disney character in 20 minutes (with admission to Disney’s Hollywood Studios. )Number of costumes: for major characters such as Minnie Mouse who has over 200 costumes and Mickey Mouse with almost 300 costumes. Regular Cast Members: require 1.8 million costume pieces to be maintained and 200,000 pieces to be constructed annually. There are 3700 different costume designs. 2.5 million Costumes exist in Costuming for Cast Members to “check out”. Each CM is allowed to have three complete costumes out at any one time. They may also request one coat and hat. This keeps many of them running between the attractions and costuming to have the right outfit for the attraction (s) they are working that week. Number of character meet and greets: 160 different characters are available for you to meet and greet, get photos and autographs with at Walt Disney World. They are not all out every day so check your Times Guide at the front of the park to check on who is out where and when. You can also ask a Cast Member. Number of food items: Of the 6000 different food items sold in the park annually-Turkey Legs sold: over 1.6 million; there are also 3.3 million Mickey Premium Ice Cream Bars sold each year; 5.8 million hot dogs and 9.7 million hamburgers; 9 million pounds of French fries; 3.2 million pizzas; This is where the big money is made, along with merchandise sales, such as the 3.5 million mouse ears sold. Number of Fireworks performances: 1,000 in a year, making WDW North Americas largest consumer of fireworks. Number of live animals: Animal Kingdom is home to 1700 animals, 5000 fish and 2000 insects; and Epcot hosts 6000 sea creatures in The Seas with Nemo and Friends. Tri-Circle D Ranch has horseback riding opportunities, and it is the home for the horses used to pull carriages, and the Main Street Trolley. Affection Section at Animal Kingdom allows you to visit and pet a couple dozen animals. Animal Kingdom Lodge has live animals that graze nearby. (Sea World Orlando reportedly has 3000 creatures. )Number of Special Events and Tours: 57 opportunities exist at Walt Disney World. You can learn how Steam Trains work and their history or maybe go on a VIP safari on the Wild Africa Trek. Maybe you would like to swim with dolphins or do some deep sea diving? Want to know what the Disney Parks look like behind the scenes? You can do that too!Thanks to Forbes.com, Disney.go.com, and disneybythenumbers.com for providing some of the facts for this piece.tweet


what is the philosophic legacy of aristotle

me. The philosophical legacy of Aristotle is vast, as it covers numerous areas of human knowledge and life.

Answers.com ® Wiki Answers ® Categories History, Politics & Society Society and Civilization Famous People Scientists Aristotle What is the philosophical legacy of Aristotle? Flag What is the philosophical legacy of Aristotle? Answered by The Wiki Answers ®Community Answers.com ® is making the world better one answer at a ti

He serves as a kind of ideal for comprehensive thought and study, given the breadth and depth of his still-influential writings on art, education, metaphysics, theology, and politics, among many other fields. Yet it is his contribution to the development of the physical sciences and of logic that perhaps stands out above all, as he can safely be said to have founded these sciences while also providing a blueprint for their development for those who have come after him. Along with Plato, Aristotle is a philosopher of whom it should be said that the world would not be the world that it presently is if he had never lived. Was this answer useful? Yes Somewhat No What is the philosophic legacy of Aristotle? No one knows what's the solutions, but they say that they will knowsoon. Is Aristotle a greek philosopher? Yes, Aristotle is a Greek philosopher. Topper1992 111 Contributions What were some of the philosophical concerns of Aristotle? The philosophical concerns of Aristotle are the arguments from the uncaused causer and the unmoved mover. What was Aristotles legacy? Aristotle studied logic and numerous amounts of psychology. He taught many students as well as taught himself. Many would agree that he was a great man. . Themendios 232 Contributions Who was the Greek philosopher who taught Aristotle? Plato was the teacher of Aristotle. Silentstar 6 Contributions What are Aristotles' philosophical ideas? Most of Aristotle's ideas were disregarded by other scientists. Hismost famous theary was that the Earth was spherical and in thecenter of the universe.33samilia33 9 Contributions Who are the philosophers socrates Plato and Aristotle? SOCRATES: Ancient Greek, changed philosophy from a study of natural science to ethics and politics. Wrote nothing. PLATO: Ancient Greek, student of Socrates, most influential …Aristotle and Socrates were philosophers of what period? Classical period The philosopher Aristotle is known as the father of what?of phycics and astronomy Were socrates Aristotle and Plato philosophers? Yes. What did philosopher Aristotle write books about?ethics Kittycocoalove 60 Contributions Was Plato a philosopher student of Aristotle? No! Student of Socrates. Answered In Aristotle What type of a philosopher was Aristotle?cow Answered In Alexander the Great Which Greek philosopher taught Aristotle? Plato. Answered In Philosophy and Philosophers What did Aristotle Greek philosopher teach? Aristotle taught that people should live lives in moderation and use reason in life Answered In Philosophy and Philosophers Were Socrates and Aristotle Roman philosophers? No Durell Hamilton 1 Contribution Answered In Philosophy and Philosophers Which philosopher taught Aristotle? Plato


how long to saute potatoes

hod For the potatoes Blanch the diced potatoes in the boiling water for 30 seconds and drain in a colander so the steam can escape. On a high heat, in a large frying pan, sauté the diced potatoes in the oil, season with salt and pepper and cook for 4-5 minutes, stirring every minute until golden brown.

Raymond Blanc's sautÃ© potatoes recipe

Raymond Blanc's sauté potatoes recipe The perfect accompaniment to a steak, these sauté potatoes are quick and easy to cook. Delicious: Raymond Blanc's sauté potatoes By Raymond Blanc12:33PM GMT 12 Feb 2013Raymond Blanc’s recipe is the perfect accompaniment to a steak or simple pan-fried pork chop and crisp sage leaves and Dijon mustard.4 medium potatoes, Maris piper or King Edward, peeled, diced (about 1cm), rinsed under cold water and patted dry.30ml (2 tbsp) rapeseed oil2 pinches sea salt2 pinches pepper, ground black Related Articles Which potato to grow26 Jul 2010Michael Caine reveals recipe for 'best roast potatoes in the world'19 Dec 2009The fight against potato blight30 Oct 2012Roux's roast potatoes recipe16 Dec 2012The best potatoes for your kitchen29 Jan 201320g butter, unsalted For the persillade:50g/1 handful parsley, flat leaf, finely chopped30g (½) shallot, medium, peeled (mix parsley and shallot together)1 clove garlic, peeled, finely chopped Met

Reduce the heat; add the butter and two-thirds of the persillade. Taste and adjust the seasoning if necessary. Reserve. Chef’s notes: Blanching the potatoes: of course you can cook the potatoes from raw, but too often they will brown too quickly as they have a high starch content. By blanching them you are neutralising the starches, giving the potato a better texture and colour. Raymond Blanc's sauté potatoes recipe Total time: 10 minutes Recipes Food and Drink »Related Partners Browse our great wine offers to go with any recipe Get £75 off your first 5 shops with Waitrose In Recipes10 tequila cocktails you need to try now Delicious pea recipes for summer Lorraine Pascale's healthy strawberry tart with lemon vanilla cream The only barbecue recipes you'll need this summer Gordon Ramsay's green juice recipe More recipes from Telegraph Food Telegraph Food Email More from the web All Auctions In One Place View lot!View lot!View lot!View lot!SEARCH ALL AUCTIONSMore from the web {phone JS}Phone Plan Finder Prev Next Loading... Deals powered by Whistle Out More from the web World News The latest world news View US News Latest and breaking stories from the United States View Film All the latest film trailers, reviews and features View Football Catch up on all the latest football news and results View


juvenile idiopathic arthritis definition

Juvenile Idiopathic Arthritis (JIA) refers to a heterogeneous group of diseases that share the common feature of arthritis of unknown origin occurring before the age of sixteen. JIA is an inflammatory disorder that primarily affects synovial joints. Each subtype has distinct clinical, immunologic and genetic features.

Juvenile idiopathic arthritis

Juvenile idiopathic arthritis Ania Van Meer Editors: Sultan Chaudhry and Eric Wong Faculty reviewer: Dr. Raj J. Carmona, Assistant Professor, Division of Rheumatology, Department of Medicine, Mc Master University Definition J Rheumatol. 2004 Feb;31 (2):390-2. Lancet. 2011 Jun 18;377 (9783):2138-49.

Together, the subtypes of JIA comprise the most common chronic rheumatic disease of childhood. To be considered JIA, onset of the disease must take place before the age of sixteen, be characterized by inflammation in at least one joint, and last for at least six weeks. Classification J Rheumatol. 2004 Feb;31 (2):390-2. Lancet. 2011 Jun 18;377 (9783):2138-49. Orthop Clin North Am. 2012 Apr;43 (2):213-25, vi. The International League of Associations for Rheumatology (ILAR) classification is the most commonly used schema, and includes 8 subtypes. ILAR sought to create mutually exclusive homogeneous subgroups under JIA for research and prognostic purposes, but challenges to classification persist. Table 1: ILAR classification of JIA (2001)Type Features Systemic JIAArthritis in 1 or more joints with or preceded by fever of at least 2 weeks’ duration that is documented to be daily for at least 3 days, and accompanied by one or more of the following features of systemic inflammation: Evanescent, erythematous rash Lymphadenopathy Hepatomegaly and/or splenomegaly Serositis Number of joints affected varies, and arthritis may develop after systemic symptoms improve. Early destructive joint change is characteristic. Oligoarthritis Arthritis affecting up to 4 joints during the first 6 months of disease. Persistent type: Affecting not more than 4 joints throughout the disease course Extended type: Extending to affect more than 4 joints after six months Polyarthritis Arthritis affecting 5 or more joints in the first 6 months of disease. RF positive: at least 2 positive tests for RF at least 3 months apart during the first 6 months of disease. RF negative Psoriatic arthritis Arthritis and psoriasis, or arthritis and at least 2 of: Dactylitis Nail pitting or onycholysis Psoriasis in a first-degree relative Enthesitis-related arthritis Arthritis and enthesitis, or arthritis or enthesitis with at least 2 of: The presence of or a history of sacroiliac joint tenderness and/or inflammatory lumbosacral pain The presence of HLA-B27 antigen Onset of arthritis in a male after age 6Acute (symptomatic) anterior uveitis History of any of the following in a first-degree relative: ankylosing spondylitis, enthesitis relatedarthritis, sacroiliitis with inflammatory bowel disease, Reiter’s syndrome, or acute anterior uveitis Undifferentiated arthritis Arthritis that fulfills criteria in no category or in 2 or more of the above categories. Classification table notes RF: Rheumatoid factor HLA: Human leukocyte antigen Dactylitis: inflammation of an entire digit (finger or toe) Serositis: inflammation of a serous membrane (pleura, pericardium, or peritoneum) Enthesitis: inflammation of an enthesis, the site where a ligament or tendon inserts into bone Pathogenesis Orthop Clin North Am. 2012 Apr;43 (2):213-25, vi. Clin Rev Allergy Immunol. 2013 Oct;45 (2):227-35. Autoimmun Rev. 2011 Jun;10 (8):482-9. Immunol Rev. 2006 Aug; 212: 8-27. Int J Inflam. 2012;2012:271569. Nelson Textbook of Pediatrics, 19e, Ch 150 Hochberg Rheumatology 5e, Ch 99 Curr Opin Rheumatol. 2011 Sep;23 (5):437-43. The triggers that initiate and progress the pathology of JIA have not been identified . As with other autoimmune conditions, both genetic susceptibility and environmental factors are implicated in producing disease. The overarching mechanism in all JIA subsets is aberrant activation of the immune system. Different components of the immune system are involved in each subtype, and additional variation may exist within subtypes. One major distinction lies in whether the aberrancy involves the innate or adaptive immune system, described as autoinflammatory and autoimmune processes, respectively. Autoinflammatory vs. autoimmune disorders In autoinflammatory processes, the innate immune system is responsible for tissue inflammation. The autoinflammatory diseases are a group of hereditary conditions that cause recurrent, unprovoked inflammation in the absence of infection (e.g. systemic JIA). Autoimmune processes involve the adaptive immune system and are based on antigen-dependent T-lymphocyte and B-cell autoantibody production. Autoimmune disease results when immune reactions are triggered by environmental factors in a genetically susceptible individual and when these reactions produce clinically significant signs and symptoms. Autoimmune diseases are often characterized by the presence of autoantibodies against autoantigens such as DNA, cell surface molecules, and intracellular proteins. Autoimmune conditions have strong associations with HLA phenotypes. Cytokine dysregulation is common to the pathogenesis of both autoinflammatory and autoimmune diseases. For example, IL-1β is the proinflammatory cytokine most involved in autoinflammatory diseases. IL-1β binds to its receptor (IL-1R type I) and thus induces the transcription of proinflammatory genes leading to inflammation and tissue damage. Disorders of innate and adaptive immunity most likely represent a spectrum of immune dysfunction, with many rheumatologic conditions being polygenic in nature. Pathogenetic mechanisms by JIA subtype Systemic arthritis (10-20% of JIA cases)Autoinflammatory disease involving a predominant abnormality of the innate immune system. The trigger is unknown. The multi-system inflammation seen in this subtype is due to complex underlying interactions of immune cells, and the host of cytokines they produce. Early in the disease process, cells of the innate immune system (monocytes, macrophages and neutrophils) are activated, and secrete pro-inflammatory cytokines and proteins (such as IL-1 β, IL-6, IL-8, IL-18, TNF-a, and macrophage colony-stimulating factor). These cytokines amplify the inflammatory process and generate clinical signs and symptoms. For example, IL-1β has been observed to produce fever, anorexia, pain hypersensitivity, joint destruction, vasculitis, and thrombosis. Serology: classically negative for RF and ANAGenetics: No association with HLA genes. Genetic studies have mainly demonstrated mild contributions from cytokine/chemokine gene polymorphisms involving the promoter elements and genes encoding IL-6 and macrophage inhibitory factor (MIF). Environment: A seasonal pattern of onset suggests a viral etiology. To date, only parvovirus B19 infection has been reported in association with the onset and exacerbation. Oligoarticular arthritis (50-60%) and polyarticular arthritis (30-35%)Despite subtle but important molecular differences in the immunopathology of oligo- and polyarticular JIA, the main concepts, presented here, are similar. Oligo-/polyarticular JIA are understood to be autoimmune diseases involving a predominant abnormality of the adaptive immune system, particularly T cells, but with emerging evidence for a component of innate immune system dysfunction. It is antigen-driven and lymphocyte-mediated; involves both inappropriate generation of inflammation, and a lack of the normal anti-inflammatory balance. Auto -antigen (s) (which are not known) in joint tissue activate autoreactive T cells (T helper cells, Th1 and Th17), which produce pro-inflammatory cytokines, namely IFN-γ and IL-17. Large numbers of autoreactive T cells can be found in the arthritic joint, clustered around antigen-presenting cells. At the same time, regulatory T cells (CD4+ and CD25+) seem to be inhibited by the milieu of inflammatory factors; TNF and 1L-6, for example, inhibit their activity. T reg cells normally function to maintain immunologic self-tolerance and negative control in both physiological and pathological immune responses. They can actively prevent the activation and effector function of autoreactive T cells that escape other mechanisms of tolerance. In JIA their function appears to be inhibited, and the inflammatory balance is disrupted in favour of inflammation. Self-tolerance to the auto-antigen is lost, and synovial inflammation results. The complex balance between pro- and anti-inflammatory mediators in the arthritic joint determines progression and outcome of disease. Serology: ANA: positive in 75-85% of oligoarticular JIA; 50-80% of RF- polyarticular, ~55% in RF+. Children who are ANA positive are at high risk of developing chronic uveitis. RF: Present in <25% of polyarticular JIA. Children with RF positivity are at risk for more extensive and aggressive disease. Genetics: Oligoarticular JIA has been consistently associated with HLA genes (HLA-A, HLA-B, and HLA-DR), though these associations are observed in girls and not boys suggesting further disease heterogeneity. Polyarticular JIA has been reported to be associated with HLA-DR4. Genetic association with HLA grants further evidence for oligo/polyarticular JIA as a disease of adaptive immunity. Other genes that may influence the onset or severity of disease include those that regulate cellular activation and cytokine responses (e.g. those encoding TNF-α, MIF, IL-6, and IL-1α). Further, c ertain cytokine alleles may predispose to upregulation of inflammatory cascades responsible for more severe articular disease. Environment: In general, infectious agents are the most important environmental factors in the development of autoimmunity. A potential mechanism is the cross-reactive immune response induced through molecular mimicry. The inflammatory response to infection may also support sensitization to auto-antigens through increasing their immunogenicity – for example, antigen-presenting cells may inadvertently prime T cells with broad specificities to the auto-antigen. Infectious agents that have been suggested as triggers include viral infections with influenza A, rubella and parvovirus B19 (though their pathogenicity is still not well proven), and bacterial infections with Mycoplasma pneumoniae and group A streptococcus. Additional risk factors include maternal cigarette smoking, psychological stress and joint trauma. Enthesitis-related arthritis (ERA)/Spondylo-arthropathies (Sp A) (10%)ERA encompasses Ankylosing Spondylitis, Reactive Arthritis, and Arthritis associated with IBD (though the term “spondyloarthropathies” also includes psoriatic arthritis when sacroiliitis or spondylitis is present). Traditionally considered to be an autoimmune disease triggered by GI or GU infections in HLA-B27 positive individuals (i.e. infection induces CD8–T-cell clones that react against bacterial antigens and auto-antigens in joint tissue). Recently, an autoinflammatory component has been suggested, with HLA-B27 playing a role in triggering the innate immune system. Specifically, the HLA-B27 molecule may be prone to misfolding within the cell’s endoplasmic reticulum, activating the innate immune system. Further, it is hypothesized that tissues exposed to either bacterial or mechanical stress (e.g. gut and synovium) are particularly vulnerable to increased immunogenicity. Repeated microtrauma of joints and entheses releases molecules like fibronectin. This activates synovial macrophages via toll-like recep- tors and other pattern-recognition molecules, which leads to the up-regulation of approximately 600 stress-related genes. A characteristic feature of Sp A is subclinical gut inflammation, with up to two-thirds of Sp A patients affected and demonstrating histological changes similar to those with Crohn’s disease. Surgical studies have demonstrated a causal link between Inflammatory Bowel Disease (IBD) and Sp A, in which patients with ulcerative colitis who underwent colectomy were cured of arthritis. IBD and Sp A are linked genetically, both involving polymorphisms of the genes coding for the IL23 receptor and the TNF ligand superfamily. These genes play important roles in mucosal and innate immune responses. Genetics: Genetics play a major role, with 80-90% of patients with ankylosing spondylitis being HLA-B27 positive. Other genetic factors have been implicated in the perpetuation of inflammation through cytokines (e.g. polymorphism in or near the TNF, IL-1 and IL-23 loci), further supporting the hypothesis that ERA involves innate immune system dysfunction. Environment: Enteric infection with certain gastrointestinal or genital pathogens can trigger reactive arthritis; environmental triggers for other forms of spondyloarthritis have not been identified. Psoriatic arthritis (Ps A) (5%)Though grouped under the same subtype, early- and late-onset Ps A have strikingly different clinical features; genetic and immunological studies point to equally different pathogenetic mechanisms. Early-onset subtype: Peak incidence age 2-3, with female predisposition. It appears to have association with HLA alleles other than B27 (e.g. HLA-DR5). It is characterized by ANA seropositivity in 50%, chronic uveitis, and shares substantial clinical features with early-onset oligo- and polyarticular JIA, therefore raising the possibility that dysfunction of the adaptive immune system plays a dominant role. Enthesitis and axial involvement are not seen. Later-onset subtype: Peak incidence age 10-12, with male predisposition. It is associated with HLA-B27 positivity, and carries additional features consistent with spondyloarthritis such as enthesitis, axial disease and subclinical gut inflammation. Because of the clinical similarity between older-onset Ps JIA and and other spondyloarthritides, innate immunologic mechanisms likely play a critical role in pathogenesis (see ERA Pathophysiology, just above). Classically ANA negative. Dacytilitis is seen in both subtypes, and thus may be caused by a combination of adaptive immunologic and innate autoinflammatory mechanisms. It follow that, even though early- and late-onset Ps A appear to have different arms of the immune system playing a dominant role, both are ultimately involved in each. Clinical features Int J Inflam. 2012;2012:271569. Kelleys Textbook of Rheumatology, 9e, Ch 106 Nelson Textbook of Pediatrics, 19e Ch 149 & 150 Adolesc Health Med Ther. 2012 Jun;2012 (3):67-74. Hochberg Rheumatology 5e, Ch 98Table 2: Clinical features of JIA subtypes Subtype Arthritis Uveitis Other Labs/serology Systemic JIASecond most common presenting symptom. Can be preceded by arthralgias and sometimes arthritis develops weeks to months following disease onset. Wrists, knees, and ankles are most commonly involved on initial presentation. Though usually asymptomatic, temporomandibular joint arthritis may be present. Chronic progressive arthritis develops in approximately 30-50% of patients. Not associated Fever – Spiking, quotidian Lymphadenopathy – 1/3 present with painless generalized lymphadenopthy. Mesenteric adenitis can be painful. Para-aortic adenopathy can be seen on radiography. Reactive changes seen in the nodes on biopsy. Rash – Salmon-pink, evanescent; morbilliform, macular, often with central clearing. Migratory; typically begins on the limbs and trunk. Appears with febrile episodes. Usually nonpruritic. Hepatosplenomegaly – 50% have splenomegaly; hepatomegaly less common. Serositis – Pericarditis most common, then pleuritis; peritonitis rare . Hb ↓WBC ↑↑ESR ↑↑CRP ↑↑ferritin ↑platelets ↑↑ (normal or ↓ in MAS)Oligoarticular JIALarge joints of the lower extremities predominantly affected. Often only a single joint is involved30%, especially if ANA positive ANA+ in 60%ESR/CRP mildly ↑Polyarticular JIARF negative: Symmetric or asymmetric; small and large joints; cervical spine; temporomandibular joint RF positive: Aggressive symmetric polyarthritis10%RF positive: Rheumatoid nodules Joint deformities (e.g. Boutonnière and Swan neck contractures)ANA+ in 40%In both RF+ or –: ESR ↑↑CRP ↑ /normalmild anemia Enthesitis-related JIAPredominantly lower limb joints affected; sometimes axial skeleton Acute anterior uveitis; associated with reactive arthritis and IBDEnthesitis – often symmetrical and involving lower extremities; most commonly patellar ligament insertion at the inferior pole of the patella, plantar fascial insertion at the calcaneus, and the Achilles tendon insertion at the calcaneus Subclinical gut inflammation45% + for HLA-B27ANA and RF –Psoriatic JIAAsymmetric arthritis of small or medium-sized joints, asymmetric arthritis involving < 5 joints. Both large (knee, ankle) and small (finger, toe) joints can be involved. May have axial involvement, particularly if HLA B27 positive.10-15%Psoriasis in 50%Dactylitis Onycholysis, nail pitting Subclinical gut inflammation ANA+ in 50%ESR ↑CRP ↑ / normalmild anemia ANA: Anti-nuclear antibody CRP: C-reactive protein ESR: Erythrocyte sedimentation rate IBD: Inflammatory bowel disease MAS: macrophage activation syndrome, a serious but rare complication that occurs in the setting of systemic-onset JIA; characterized by the rapid development of fever, rash, and encephalopathy; rapid rise of liver transaminase levels; disseminated intravascular coagulopathy; neutropenia; thrombocytopenia; increased triglycerides; low albumin; and a low ESR associated with hypofibrinogenemia. Mortality has been reported as high as 50%, and prompt treatment with corticosteroids is necessary and, if poor response, also with etoposideor anti-thymocyte globulin and cyclosporine. Pathophysiology Hochberg Rheumatology 5e, Ch 99 Nelson Textbook of Pediatrics 19e, Ch 621 Br J Ophthalmol. 2012 Jun;96 (6):852-6. Curr Opin Ophthalmol. 2010 Nov;21 (6):468-72. Arthritis Defined by intra-articular swelling or the presence of 2 or more of the following: Limitation in range of motion Tenderness or pain on motion Increased heat or erythema The hallmark of arthritis in JIA is inflammation of the synovial lining of the joint, which causes the above signs and symptoms. If uncontrolled, it may lead to joint destruction through progressive erosion of articular cartilage and contiguous bone. Inflammatory synovitis is characterized pathologically by: Villous hypertrophy and hyperplasia Hyperemia and edema of the synovial tissue Hyperplasia of synoviocytes Infiltration of inflammatory cells ( lymphocytes, plasma cells, macrophages, and dendritic cells)Pannus formation One of the hallmarks of JIA pathology, especially RF positive polyarticular JIA, pannus is a tumor-like expansion of inflamed synovial tissue which causes much of the joint damage. It spreads over the synovial space, degrades intra-articular cartilage and may lead to anklyosis (fusion of tissues and consequent stiffening). Pannus is caused through synoviocyte proliferation and invasion of the synovial tissue by inflammatory cells recruited from the peripheral circulation. Uveitis The uveal tract consists of the inner vascular coat of the eye, and includes the iris, ciliary body, and choroids. It is vulnerable to systemic inflammatory diseases. Inflammation may affect any one aspect of the uveal tract preferentially or all parts together.70% of patients have bilateral involvement Most common in oligoarthritis and ANA positive patients Approximately 30–70% of patients develop ocular complications. The main causes of poor vision are cataracts, band keratopathy and glaucoma. Table 3: Uveitis in JIALocation of Inflammation Anterior Uveitis (Iritis)Intermediate Uveitis (Iridocyclitis)Posterior Uveitis (Choroiditis/Chorioretinitis)Panuveitis (Panophthalmitis)% of all JIA types83%9%1%7%Signs and symptoms Pain, photophobia, and lacrimation. Conjunctival hyperemia. Rare to have pain, photophobia, or conjunctival hyperemia. Inflammation may not be noticed until severe and irreversible damage has occurred. Routine screening is therefore necessary. Blurred vision. Results in atrophic chorioretinal scarring, often with visual impairment. Secondary complications include retinal detachment, glaucoma, and phthisis. Severe pain, suppurative, marked congestion of the eye, inflammation of the adjacent orbital tissues and eyelids, and loss of vision. Long term consequences Long-term consequences of childhood-onset arthritis include: Local growth disturbances and limb-length discrepancy: Inflammation initially accelerates linear growth; continued inflammation stimulates premature closure of the growth plate, resulting in shortened bones. Generalized growth disturbance: Generalized growth disturbance – delayed growth and low weight – may occur in children with active disease and is related to active disease intensity and duration. Height may return to normal in 2 or 3 years after quiescence of the disease, as long as premature epiphyseal closure has not occurred. The etiology of generalized growth disturbance is unclear, but it is tightly linked to the degree of overall inflammation. Osteopenia: Active disease, poor nutrition (i.e. low calcium and vitamin D intake), medications such as corticosteroids, lack of exercise, and delayed menarche can all affect the peak bone mass attained in adolescence Functional disability: Including unemployment, pain and decreased overall quality of life. Treatment Adolesc Health Med Ther. 2012 Jun;2012 (3):67-74. BMC Pediatr. 2012 Mar 15;12:29. Arthritis Care Res (Hoboken). 2011 Apr; 63 ( 4 ):465-82. The treatment approach to JIA has shifted from a pyramidal approach to early aggressive therapy. Risk stratification guides optimal management, and thus treatment is individualized according to subtype of JIA, and severity of disease as measured by affected joint count, systemic features, and poor prognostic features. General approach to treatment involves anti-inflammatory and immunomodulatory medications in combination with physical and occupational therapy, an occasional need for surgery, nutritional support, and psychosocial and educational partnership with patients and parents. Table 4: Common medications used in the treatment of JIAMedication Mechanism of action Non-steroidal anti-inflammatory drugs (NSAIDs)Inhibit the enzyme cyclooxygenase (nonselective inhibitors inhibit both COX-1 and COX-2 isoenzymes), preventing prostaglandin production. Intra-articular glucocorticoids Believed to act by altering transcription; reduce numbers of inflammatory cells, and thus inflammatory mediators Non-Biologic Disease-Modifying Anti-Rheumatic Drugs (DMARDs)Methotrexate Folate antimetabolite; inhibits DNA synthesis, repair and cellular replication Biologic DMARDs Etanercept Anti-TNF-a Adalimumab Anti-TNF-a Infliximab Anti-TNF-a Abatacept Selective T-cell co-stimulator inhibitor Anakinra Anti IL-1Tocilizumab Anti IL-6Summary of ACR 2011 recommendations NSAIDs are first-line treatment for oligoarticular arthritis, often with intra-articular glucocorticoids. Methotrexate is used next, if treatment response is inadequate, or if 5 or more joints are involved (in addition to an NSAID and joint injections). TNF-a inhibitors are added if disease activity remains moderate or high. If one TNF-a inhibitor is not sufficient to adequately decrease disease activity, a second TNF-a inhibitor or Abatecept is added. Anakinra is used only in Systemic Arthritis, alongside systemic glucocorticoids for systemic features, and methotrexate and TNF-a inhibitors for active arthritis. Medical therapy NSAIDs have not been shown to alter the disease course or prevent joint damage, and are used instead to treat pain, stiffness, in addition to fever in systemic JIA. No individual NSAID has been shown to have a clear advantage over others in treating arthritis. NSAIDs commonly used are tolmetin, naproxen, ibuprofen, meloxicam, and celecoxib. Because of the various undesirable side effects of corticosteroids, particularly with respect to bone and growth, the use of systemic corticosteroids to treat arthritis is minimized. They may, however, be used as a bridging medication until other DMARDs take effect. The main indications for the use of systemic corticosteroids are severe fever, serositis, and the macrophage activation syndrome in patients with systemic JIA. Intra-articular corticosteroid injections have demonstrated efficacy in patients with oligoarthritis. DMARDs have demonstrated superior efficacy, and have been used increasingly in the treatment of JIA. DMARDs interfere directly with immune cells or their function to reduce inflammation. They are classified as either biologic (created by biologic processes) or non-biologic (synthetic). Treatment of uveitis: Corticosteroids remain the mainstay of initial treatment for uveitis. For patients requiring chronic therapy, the initial agents of choice are typically antimetabolites including methotrexate, mycophenolate mofetil and azathioprine.


what does the chief administrator do

What Is a Chief Administrative Officer? A chief administrative officer (CAO)--also known as chief operating officer (COO), top administrative executive or vice president of administration--provides the overall direction to an organization through planning, policy-making and coordinating a variety of activities.

What Is a Chief Administrative Officer?

What Is a Chief Administrative Officer? Research what it takes to become a chief administrative officer. Learn about the duties of this job, the education requirements and the salary range to find out if this is the career for you. Schools offering Business degrees can also be found in these popular choices .

They create goals, policies and procedures as well as manage the finances of the organization. They take care of contracts and agreements and find ways to keep costs down. Chief administrative officers also try to find ways to improve the organization. The following chart provides an overview of the education, job outlook and average salary in this field. Degree Required Bachelor's degree at minimum; many hold a Master of Business Administration (MBA)Education Field of Study Business administration, law, communication, economics, finance, leadership, organizational theory, operation management, information systems, human relations, marketing Key Responsibilities Keep the organization profitable and in alignment with its policies and goals; ensure that all operations run smoothly; provide leadership for personnel; prepare and oversee budgets; use good judgment in negotiating business deals Job Growth (2014-24) -1% for all types of chief executives*Mean Annual Salary (2015) $185,850 for all chief executives*Source: *U. S. Bureau of Labor Statistics What Are These Titles Other Than Chief Administrative Officer? Executive titles are often interchangeable because every company creates its own hierarchy and positions. The chief administrative officer position may also be referred to as the chief operating officer, top administrative executive or vice president of administration. In most cases the job duties are the same. What Would My Duties Be? As one of the top administrative positions in a company, you'll oversee the daily activities of employees and operations. You may be in charge of hiring or firing employees, completing performance evaluations, developing policies, negotiating contracts and delegating tasks. Depending on the size of the company, you may supervise and delegate jobs to the heads of departments. You'll likely meet with the CEO at least once a week to report on the happenings of the company. In this instance, you may be the middle-man or -woman between the department heads and the CEO. What Should I Study in School? Salary.com states that 40% of polled CAOs held a bachelor's degree, while 30% held a master's degree, as of February 2017. The U. S. Bureau of Labor Statistics (BLS) suggests earning a bachelor's or master's degree in business administration ( www.bls.gov ). However, your degree choices will depend on your field of interest and prospective employers' needs, since CAOs often advance into the position after acquiring experience in a specific industry or company. To give yourself more options, you'll want to take a program heavy in business classes. Courses should teach you accounting, project management, human relations, marketing and organizational theory. Other helpful topics include statistics, finance, operations management, economics and entrepreneurship. You'll also want to develop your decision-making, problem-solving, communication and writing skills. How Much Can I Make? The BLS reported that all types of top executives, including business executives, would see an employment decline of 1 percent for the period between 2014 and 2024. However, job prospects would depend on the industry you choose. For example, the expected demand for healthcare workers will create openings for executives in that field. Other industries may see a loss in the number of job openings, as overall demand for those services decreases. The BLS pointed to the manufacturing industry as one segment that would see a reduced need for executives. In May 2015, the average annual salary for chief executives in general was $185,850, according to the BLS. Pay Scale.com reported in October 2016 that most chief administrative officers, the 10th to 90th percentiles, earned a base annual salary between $51,234 and $203,418. When bonuses and profit sharing options were factored into the salaries, Pay Scale.com reported salary ranges between $49,473 and $234,981. What Are Some Related Alternative Careers? There are several other career options that are similar to a chief administrative officer that also require a bachelor's degree. One option is an administrative service manager. In this position, you would keep up facilities and oversee various activities including recordkeeping and office procedures. Financial management is another option. Financial managers take care of the finances in a company, through keeping up with reports, investing and other strategies to focus on long-term growth. Sales managers are also similar to chief administrative officers. They focus on the sales of an organization with the use of data, goal setting and training. To continue researching, browse degree options below for course curriculum, prerequisites and financial aid information. Or, learn more about the subject by reading the related articles below:1. Degree Options: Business Business Management Management View All Degree Options2. More Articles Administrative Certificate or License Facts Probation Officer College Courses and Training What Does a Certified Administrative Manager Do?


trivex lenses cost

Trivex lenses ($200 for single vision, $400 for progressives) are more scratch-resistant than either CR-39 or polycarbonate lenses. They can be useful if you wear rimless or semi-rimless frames, or if you’re hard on glasses.

How to Get the Best Eyeglass Lenses

How to Get the Best Eyeglass Lenses Polycarbonate or high index? Bifocals or progressives? How to pick what's right for you and save money. By Consumer Reports Last updated: December 29, 2016Picking frames that look right is tough enough.

After that all-important decision, you still have to choose eyeglass lenses and coatings. Those decisions are key to how well you’ll see and how much you’ll spend. Americans shell out an average of $275 after insurance for new glasses, and most of that money is for lenses—not frames.

You can’t always rely on salespeople, who may work on commission, to guide you. But Consumer Reports' expert steps will he

lp. Looking at Lenses The two best-selling eyeglass lenses are the most basic ones: CR-39 and the polycarbonate, both plastic. (Few people now use glass, which is heavy and breakable.) If you have a single-vision prescription (glasses to see far away or close up), you can generally get by with CR-39 lenses.

They can be inexpensive—we found them for $29 to $149—but they can look thick with stronger prescriptions. A more durable, thinner, lighter, and more popular option is polycarbonate lenses, which we found for $9 to $205. Some retailers even offer lenses free of charge as part of packages. But if you need glasses to see both near and far, lens choices get more complex. Here are four, along with their national average costs: Progressives ($260) provide a smooth, gradual change in lens strength for seeing well at any distance. Consider them if you need glasses for distance and reading and find the split screen of bifocals or trifocals uncomfortable.

Pricier than bifocals ($105), they can be made with CR-39, polycarbonate, or high-index lenses. High-index lenses ($150 for single vision, $350 for progressives) are thinner and lighter than CR-39 or polycarbonate lenses, and they will work for even the strongest prescriptions.

High-definition lenses ($310 for progressive HD lenses) offer sharper vision and better peripheral vision than standard technology. You might want to opt for them if you have more complex visual problems, such as cataracts or corneal scars. They can be made with CR-39, polycarbonate, or high-index lenses.

More on Buying Eyeglasses How to Avoid Being Gouged When Buying Eyeglasses Your Guide to Buying Glasses Online8 Great Ways to Save on the Cost of Eyeglasses Coatings to Consider Lens coatings are meant to protect your eyes from light or increase lens durability. Five common treatments to know about: Anti-scratch —generally a good idea for all—comes with 95 percent of plastic lenses. Check the warranty; retailers such as Warby Parker will replace lenses that get scratched in the first year after purchase free of charge. Anti-reflective coating ($50 to $100), often bundled with high-index and HD lenses, used to be hard to clean and smudge-prone but now has anti-smudge/anti-fog technology. If you have trouble seeing properly when on a computer, driving, and at night, consider them. Ultraviolet protection ($20 to $100) is a good idea for most people because the sun’s UV rays may boost the risk of cataracts. Most lenses already come with this coating; make sure yours do. Photochromic coating ($50 to $150) darkens in sunlight and shields you from UV rays. It’s helpful if you’d rather not carry separate sunglasses. Blue-light-blocking coatings ($30 to $180) are said to reduce exposure to computer screens’ LED light. (Some studies suggest that overexposure can damage the retina and increase the risk of age-related macular degeneration and cataracts . )But there’s “no strong evidence that blue light affects the retina in any way we have to be worried about,” says Neil Bressler, M. D., chief of the retina division at the Johns Hopkins Wilmer Eye Institute. The orange or yellow tints may give you the perception that the tint is soothing to your eyes when you’re on a computer, but there is no strong evidence that you need them for eye health or safety.4 Ways to Save Money Ask your optician for a discount. “Prices are not set in stone,” says Steve Kodey, senior director of industry research for The Vision Council, a trade organization. Have costs broken down. Eyeglass lenses and coatings are often bundled together. A listing of prices will help you see where you can shave costs. Find out about cheaper alternatives. Some lenses and coatings are available in less expensive generic forms. Check online prices. Kodey says that many optical shops are inclined to match those prices. If not, check the big-box stores. At Costco, a pair of HD progressive lenses with anti-reflective coating and UV protection costs $130; at Walmart, the price is $255. Do you know which lenses are in your glasses? Tell us more about it below.40Eyeglass stores in Our Ratings. Current Eyeglass store Ratings


what is what is the fastest car in the world

The Bugatti Chiron is currently the fastest car in the world 2017 with a top speed of 288 mph (464 Km/h). It could even go faster but it’s electronically programmed to not go over the 288 mph limit.

Top 10 Fastest Cars In The World 2016-2017Luxury Cars Feb 1, 20175 points Emil Anton Follow 32Top 10 Fastest Cars In The World 2016-2017If you were wondering which is the world’s fastest car, you’re in luck, because we’re about to take a look at the fastest cars in the world 2017!

Although we have countless ways to measure a car’s performance, we’re, of course, going to go for the top speed. This is a top 10 of the Fastest Cars In The World as of the end of 2016 and beginning of 2017 based solely on the top speed a production car can reach.

The problem with finding the world’s fastest production car is the fact that it is a “production car” because there’s a very thin line from in-line car to super-race-car. Here is a list of the 10 fastest cars in the world. The following are all street legal production cars, which have speeds that have been (mostly) verified in some one shape or another. That means no SSC Tuatara, at least not yet, but it does merit a mention in this ranking. Top 10 Fastest Cars In The World 20

17:#10 Mc Laren F1 – Top speed: 243 mph/386 kph. Base price: 1 million dollars… but good luck with that. Since there were only a very few made, car collectors have been fighting each other for them. Just recently one Mc Laren F1 was sold at auction for 13.5 million dollars making it one of the most expensive cars in the world.

Apparently the owner is driving it around town.#9 Koenigsegg CCX ~ Top Speed: 245 mph/394 kph. Price tag: 4.8 million dollars. Only 3 Trevita cars were produced.#8 Saleen S7 Twin – Top Speed: 248 mph/399 kph. Price tag: $500,000#7 SSC Ultimate Aero TT – Top Speed: 256 mph/412 kph. When released this was the crown jewel of the Shelby Company. Price tag: $750,000#6 Koenigsegg Agera R – Top speed 260 mph/418 kph. Price tag: 1.6 million dollars. Despite having a theoretical top speed of 273 miles per hour, there isn’t any official recording over 260.#5 Bugatti Veyron Super Sport – Top Speed: 267 mph/430 kph. Price tag: 1.9 million dollars.#4 Hennessey Venom GT ~ Top Speed 270 mph/ 435 kmh Price tag: $1.2 million#3 SSC Tuatara 2017 – Top Speed: 276 mph/444 kph. This is the follow-up from the Ultimate Aero, that’s been in development since 2011. Price tag: $1.3 million#2 Koenigsegg One 1 – Technical top speed: 280 mph/450 kph. Base price: $2.8 million Towards the end of last year, they made a special car to mark the sale of their hundred piece, named Hundra, that sold for: 4.2 million dollars in Singapore.#1 Bugatti Chiron ~ Top Speed 288 mph/ 464 kmh

If you’d like to know more about the world’s fastest car in terms of top speed, you can read these 10 things you need to know about the Bugatti Chiron!This was a quick top 10 of the Fastest Cars In The World! Hope you guys enjoyed it and as usual if you have any questions leave them in the comments bellow and we’ll do our best to answer them!Enjoy this amazing video!Top 10 Fastest Cars In The World 2017Your browser does not currently recognize any of the video formats available. Click here to visit our frequently asked questions about HTML5 video.0:00 / 6:09Or check out this amazing race: That ends our article on these luxurious automobiles and with 2016 just around the corner we look forward to a new top and a new video! Stay tuned!Share Next post Cars fastest cars luxury supercars top 10 world Discussion - 3 Responses Osbert Chan says: Oct 21, 2016 at 10:25 pm Another mistake, the Koenigsegg Agera R’s picture is actually a Koenigsegg One:11 Login to vote Log in to Reply Aadil says: Jul 29, 2016 at 9:23 am There is a small mistake in this because the second rank goes to koenigsegg one 1 due to its top speed is 273 MPH / 439 KPH1 Login to vote Log in to Reply Rykardo says: Jan 19, 2018 at 3:48 pm Sorry, but the Koenigsegg Agera RS is, currently, the World’s fastest production car, with a maximum speed of 284mph. But nice try – can’t you use Google?0 Login to vote Log in to Reply Submit reply You must be logged in to post a comment.


what color backs the military

With the establishment of yellow for the Armor and the use of green for the insignia on the Armor flag, the colors for the Military Police were reversed. The current colors, green piped with yellow, were assigned by regulation 600-60-1 dated 26 October 1951.

Service Arms Branch Primary Color Piping Color Notes Adjutant Generals Dark Blue 65012 cloth 67126 yarn PMS 539 #003049Scarlet 65006 cloth 67111 yarn PMS 200 #c41e3a The pompons on the Adjutant Generals' caps were topped with white in 1851. The facings were listed in the specification for the Adjutant General's uniform in September 1915 as dark blue. In Circular number 70 dated 28 October 1936, the Adjutant General's Corps and the National Guard Bureau exchanged colors and the present colors were established for the Adjutant General's Corps. The blue used in the branch insignia is ultramarine blue rather than the branch color. Army Security (Obsolete)Teal Blue and White

The Army Security branch USAR was merged with the newly established Army Intelligence and Security Branch on 1 Jul 62. The branch was subsequently redesignated to the Military Intelligence Branch on 1 Jul 1967.

Chaplain Corps Black cloth 65018 yarn 67138NO PIPINGChaplains have used black since 1835. In regulations dated that year, a black coat was prescribed for Chaplains. Civil Affairs Purple 65009 cloth 67115 yarn PMS 267 #4f3595White 65005 cloth 67101 yarn PMS white. The colors were approved for civil affairs units in June 1956. Corps of Engineers Scarlet 65006 cloth 67111 yarn PMS 200 #c41e3a White 65005 cloth 67101 yarn PMS white. Scarlet and white were established as the Corps of Engineers colors in 1872. Before that date, several other colors had been associated with the Engineers. Finance Corps Silver Gray 65008 cloth 67137 yarn PMS 422 #afaaa3Golden yellow 65001 cloth 67104 yarn PMS 116 #fcd116Silver gray piped with golden yellow was prescribed for the Finance Corps in 1920. Command Sergeant Major Teal Blue and Yellow. Colors do not vary; is branch immaterial. Inspector General Dark Blue 65012 cloth 67126 yarn PMS 539 #003049Light blue 65014 cloth 67120 yarn PMS 5415 #607c8c The Inspector General's Department in 1851 had pompons of buff with upper one third in scarlet. In 1915, specifications established the facings as dark blue. The 14 October 1921 regulation established the colors as dark blue piped with white. Circular #70, dated 18 October 1936 announced the exchange of colors with the Judge Advocate General's Department that resulted in the present colors. Insular Affairs (1902-1939)Dark Blue 65012 cloth 67126 yarn PMS 539 #003049The Division of Customs and Insular Affairs was organized on 13 December 1898. This Division was responsible for the administration of U. S. possessions and islands under military occupation. The War Department managed some of the possessions and the State Department managed some of the affairs. In 1902, the Bureau was established with the War Department responsible for all of the possessions. The insignia was authorized on 31 December 1902 in General Regulations and amended by General Orders No. 132 on the same date. The Bureau was transferred from the War Department to the Department of Interior in 1939. JAG Corps Dark Blue 65012 cloth 67126 yarn PMS 539 #003049White 65005 cloth 67101 yarn PMS white. In 1851, the pompon for the Judge Advocate was prescribed as all white. Specifications in 1915 indicated that the facings of the Judge Advocate General were dark blue. AR 600-35, October 1921 assigned dark blue piped with light blue to the Judge Advocate General. Circular 70, 28 Oct 1936, announced the exchange of colors of the Inspector General's Department with the Judge Advocate General's Department so that dark blue and white were adopted on that date for the Judge Advocate General. Logistics Soldier Red 80095 cloth 67157 yarn PMS 209 #75263d Medical Department Maroon 65017 cloth 67114 yarn PMS 504 #511e26White 65005 cloth 67101 yarn PMS white. Green was prescribed as the first Medical Department color in 1847 when the sash for Medical Officers was described. The green was established in the insignia of the Hospital Stewards uniform on 31 October 1851 and in 1857 the green was piped with yellow and the pompon was topped with medium or emerald green. Later the pompon was green piped with white until 1902 when the maroon color was adopted. In 1903, the Hospital Corps chevrons were maroon piped with white. Maroon and white were established for all branches of the Medical Department by the uniform specifications dated October 1916. Military Intelligence Oriental Blue 80176 cloth 67172 yarn PMS 285 #3a75c4Silver Gray 65008 cloth 67137 yarn PMS 422 #afaaa3DA GO 38 dated 3 July 1962 announced the establishment of the Army Intelligence and Security Branch. In October 1962, DCSPER approved the colors for the branch. The branch was redesignated as the Military Intelligence Corps by GO 25 dated 16 June 1967. The same colors were used when the name was changed. Military Intelligence (Obsolete)Golden Yellow and Purple Military Police (1922-1951)Yellow 65002 cloth 67108 yarn PMS 123 #ffc61e Green 65007 cloth 67129 yarn PMS 357 #215b33The color yellow piped with green was assigned to the Military Police by AR 600-35 dated 20 April 1922. Military Police (1951-Present)Green 65007 cloth 67129 yarn PMS 357 #215b33Yellow 65002 cloth 67108 yarn PMS 123 #ffc61e

National Guard Bureau Dark Blue 65012 cloth 67126 yarn PMS 539 #003049Ordnance Corps Crimson 65013 cloth 67112 yarn PMS 220 #aa004f Yellow 65002 cloth 67108 yarn PMS 123 #ffc61e In 1835, the Ordnance Corps had a red plume - the same as Artillery. Crimson was prescribed as the Ordnance color in 1851. In 1902, it was changed to black and scarlet. Crimson and yellow were established as the branch colors on 14 October 1921. Psychological Operations Bottle Green 65025 cloth 67191 yarn PMS 343 #00563f Silver Gray 65008 cloth 67137 yarn PMS 422 #afaaa3The colors were traditionally used on the Psychological Operations flags and guidons. Public Affairs Since the public affairs functions are multi-branch, teal blue (branch unassigned) is the color used on the plaque and guidons. Yellow is used as the secondary color for the insignia and numbers on guidons. Quartermaster Corps Buff 65015 cloth 67135 yarn PMS 465 #c1a875The plumes for the Quartermasters in 1835 were prescribed to be light blue. In 1851, the pompon on the caps of all members of the staff corps consisted of the lower two-thirds in buff and the upper third of the color of the corps. Light or saxony blue was used for the Quartermaster Department. In 1884, the color buff was adopted which is presently used. Light blue is still used as the secondary color on flags for Quartermaster units. Signal Corps Orange 65004 cloth 67110 yarn PMS 1655 #f95602White 65005 cloth 67101 yarn PMS white Orange was selected in 1872 as the Signal Corps branch color. In 1902, the white piping was added to conform to the custom that prevailed of having piping of a different color for all branches except the line branches. Staff Specialist Green 65007 cloth 67129 yarn PMS 357 #215b33Transportation Corps Brick Red 65020 cloth 67113 yarn PMS 202 #8c2633Golden Yellow 65001 cloth 67104 yarn PMS 116 #fcd116During World War I, the personnel of the Motor Transport Corps wore purple piping on overseas caps. In 1919, the Transportation Corps was assigned the colors scarlet piped with green. Under the requirement of an Act of Congress of 1920, these services were combined under the Quartermaster Corps. When the Transportation Corps was established in 1942, the brick red piped with golden yellow was assigned as the branch color. Warrant Officers (Obsolete)Brown NONEBrown has been used as the color to represent warrant officers Women's Army Corps (1942-1978)Mosstone Green and Old Gold


what are the requirements for bethune cookman

Bethune-Cookman is a historically black college found in Daytona Beach, FL. Nearly three thousand students are enrolled at Bethune-Cookman University. Most of the students accepted into Bethune-Cookman University earned an SAT score within the range of 730 - 910, or got accepted with an ACT composite score in the range of 15 - 18. We were able to estimate the Bethune-Cookman average GPA (2.64 - 2.81) of admitted students by applying sampled GPA data on over 150 schools.

Bethune-Cookman University (Bethune-Cookman)

Get more information on Bethune-Cookman University: Requirements & Admissions Tuition & Costs Programs Available Financial Aid Eligibility Student Services Offered

Bethune-Cookman accepts about 70 percent of those that apply per year. Of those that are accepted, roughly 21 percent continue on to register and enroll. You can get more information from the admissions office website at cookman.edu. Overall Bethune-Cookman Acceptance Rate - Fall 201366.6%The overall acceptance rate for Bethune-Cookman University was reported as 66.6% in Fall 2013 with over 7,430 applications submitted to Bethune-Cookman. Both in state and out of state applicants are included in these figures. We do not have data on transfer acceptance rates currently. Accepted Applicants Profile Bethune-Cookman ACT Scores: 15 - 18Bethune-Cookman Average GPA: 2.64 - 2.81How Hard Is It to Get into Bethune-Cookman University? Applicant Selectivity: Low When considering the average GPA and SAT/ACT scores of students accepted to Bethune-Cookman University and comparing to other schools nationwide, applicant selectivity is lower than most other schools. Historical Trend and Acceptance Rate 2018 Projection Year Acceptance Rate2009 69.12010 68.72011 672012 73.52013 66.6The historical trend chart shows acceptance rates from the previous years, from which we have projected the acceptance rate for the 2018-2019 school year. The overall acceptance rate trend for Bethune-Cookman has been staying about the same from year to year. Acceptance Rate By Year2009-2010: 69.1%2010-2011: 68.7%2011-2012: 67.0%2012-2013: 73.5%2013-2014: 66.6%Projected Bethune-Cookman Acceptance Rate 2018-2019: 69.0%How Does Bethune-Cookman Compare?66.6%This School55.5%National Avg The acceptance rate for Bethune-Cookman University is twenty percent higher than the national average of all colleges which is around 55.5%. Nearby Similar College Acceptance Rates School Acceptance Rate Stetson University 59.3%Rollins College 58.9%Adventist University of Health Sciences 13.4%Flagler College-St Augustine 49.9%Florida Christian College 41.7%Admissions Rates & Enrollment Rates - Details General Recommendations & Requirements for Admissions High School GPA Scores are Required High School Class Rankings are Recommended School Records are Required College Prep Classes are Recommended Submit Recommendation Letters are Required Standardized Test Scores (SAT,ACT,...) are Required TOEFL Exam (Foreign Language Students) are Required Number of Applicants, Percent Accepted, and Enrolled Students (Fall 2013)Total Male Female Applicants 7,429 36.1% 63.9%Admitted 4,948 34.7% 65.3%Enrolled 1,022 41.0% 59.0%Average ACT/SAT Scores of Applicants to Bethune-Cookman (Fall 2013)ACT ® Score Range English 14 - 18Math 15 - 18Composite Score 15 - 18To learn more about the data and calculations that power this site, please visit our data discussion page.


the meaning of stinchcombe

Definition: Stinchcombe is an ancient Gloucestershire surname evidently derived in the first place from the parish of that name in the county. There was a yeoman thus called in Hawkesbury 200 years ago; and last century the name occurred in Acton and Cromhall (Bigl.

"Stinchcombe Surname Meaning & Statistics505,406 thmost commonsurname in the world Approximately 469people bear this surname Most prevalent in: England Highest density in: Canada

Read More About This Surname We found records about Stinchcombe 4,902 Birth Records 1,880 Marriage Records1,888 Death Records 3,106 Census Records53 Military Records 44 Immigration Records495 Newspaper Articles Show all records Stinchcombe Surname Distribution World Nations2014By incidence Save Fullscreen Highcharts © Natural Earth2014 1901 1881 1880Country Incidence Frequency Rank in Nation England 177 1: 305,085 26,770Canada 139 1: 254,874 24,984Australia 76 1: 310,082 30,024United States 62 1: 5,165,648 268,319New Zealand 7 1: 649,536 38,459Wales 3 1: 1,033,333 9,097Malaysia 1 1: 30,233,000 40,340The Bahamas 1 1: 351,461 2,613Japan 1 1: 127,100,000 29,046France 1 1: 65,959,000 277,603Show All Nations Stinchcombe Surname Meaning Submit Information on This Surname for a Chance to Win a $100 Genealogy DNA Test DNA test information Descriptive Writing introductory reference Stinchcombe is an ancient Gloucestershire surname evidently derived in the first place from the parish of that name in the county. There was a yeoman thus called in Hawkesbury 200 years ago; and last century the name occurred in Acton and Cromhall (Bigl.). There are still Stinchcombes in Cromhall and Hawkesbury.— Homes of Family Names in Great Britain (1890) by Henry Brougham Guppy A parish in Gloucestershire.— Patronymica Britannica, written: 1838-1860 by Mark Antony Lower Stinchcombe Surname Variants & Similar Names Similar Surnames Surname Similarity Incidence Prevalency Stinchecombe 96 1 /Stinchcomb 95 1488 /Stenchcombe 91 - -Stynchcombe 91 4 /Stinchicomb 91 - -Stynchcomb 86 12 /Stunchcomb 86 1 /Stinchcamb 86 1 /Stenchcomb 86 - -Stenchicomb 82 - -Stinchcombe Reference & Research Stinchcombe Family Tree DNA Project - A description of a group researching the paternal lines of men who bear the surname with the help of DNA analysis. Click here to submit links to web-pages detailing the Stinchcombe family. The surname statistics are still in development, sign up for information on more maps and data By signing up to the mailing list you will only receive emails specifically about surname reference on Forebears and your information will not be distributed to 3rd parties. Footnotes Language Description: Descriptions are largely reproduced from 3rd party sources; diligence is advised on accepting their validity Surname distribution statistics are generated from a global sample of 1.6 billion people Rank: Surnames are ranked by incidence using the ordinal ranking method; the surname that occurs the most is assigned a rank of 1; surnames that occur less frequently receive an incremented rank; if two or more surnames occur the same number of times they are assigned the same rank and successive rank is incremented by the total preceeding surnames Similar: Surnames listed in the ""Similar Surnames"" section are phonetically similar and may not have any relation to Stinchcombe "


where is wild creek reservoir

nia Wild Creek Reservoir is a reservoir located just 7 miles from Palmerton, in Carbon County, in the state of Pennsylvania, United States, near Trachsville, PA. For Fishing License purchase, fishing rules, and fishing regulations please visit Pennsylvania Fish & Wildlife

"Wild Creek Reservoir Fishing near Palmerton, Pennsylvania Write a Report Summary Fishing Times Fishing Reports Hot Spots Nearby Guides Courtesy Google Images Read Fishing Reports Details for Wild Creek Reservoir Species Caught Here: Unknown Access: Public Property Body of Water Type: Reservoir Lat/Long: 40.896202 -75.562965 Edit Details Bodies of Water near Wild Creek Reservoir Wild Creek Dam Palmerton, PAWild Creek Dam Jim Thorpe, PAWild Creek Palmerton, PABauer Creek Palmerton, PAPenn Forest Reservoir Brodheadsville, PAAll Carbon County Fishing Spots Description for Wild Creek Reservoir, Carbon County, Pennsylva

. Please remember to check with the local Fish and Wildlife department to ensure the stream is open to the public. Now get out there and fish! Check out our Fishing Times chart to determine when the fish will be most active. Fishing Hot Spots near Wild Creek Reservoir""If you live nearby - you must go and fish! But to drive 130 miles to get zero? But it all depends..."" Spruce Run""It's all depends on God Will. One year Saxton falls prime spot, other year Point Mt rd in 2011 PMR..."" Musconetcong River""My cousin's best bullhead so far!..."" Pohatcong Creek View All | Driving Directions Local Hotel Rooms City Check In Check Out Guests Rooms Check Rates Best Fishing Times near Wild Creek Reservoir Best Fishing Times: 12:00AM to 12:00AM, and 12:00AM to 12:00AMAlternate Fishing Times: 12:00AM to 12:00AM, and 12:00AM to 12:00AMFishing Wild Creek Reservoir, PA on 4/10/2018 will be best from 12:00AM through 12:00AM, and from 12:00AM to 12:00AM. If those times won’t work for you, 12:00AM to 12:00AM looks promising , as does 12:00AM to 12:00AM Remember to adjust these times based on barometric pressure, and weather changes. Guides, Charters, and Shops near Wild Creek Reservoir Border Water Outfitters Bill's Guide Service Lake Wallenpaupack L. D. Guide Service Lackawaxen River Fishing Tours Lackawaxen River Backwoods Angler Schuylkill River Great Escape Charter Fishing View All "


effects of hyperthyroidism

Hyperthyroidism effects can be serious, causing long-term health complications for patients who go without treatment. Hyperthyroidism results when the body produces too many thyroid hormones. This elevates the body’s metabolic rate, causing symptoms such as a rapid heartbeat, inability to sleep, and hand tremors, according to Healthline.

"Home | Fast Features Tags: Thyroid Disorders | thyroid | disease | long-term | effects | hyperthyroidism Thyroid Disease: Long-Term Effects of Hyperthyroidism By Breana Noble | Tuesday, 16 Feb 2016 06:43 PMEmail Article | Contact | Print | A ATweet

Hyperthyroidism doesn't always worsen without treatment, but long-term effects of nontreatment should be considered. EDITOR'S NOTE: The One Secret to a Healthy Thyroid

Some of the most serious effects of hyperthyroidism impact the heart’s function. Rapid heart rate, atrial fibrillation (which occurs when the heart beat is abnormal), and congestive heart failure (when the body is unable to circulate the blood) may occur, according to the Mayo Clinic.

Treatment may reverse the conditions, though. Other cardiovascular diseases linked to long-term effects of hyperthyroidism include hypertension and cardiac arrest, Endocrine Web reported. Since too many hormones may harm the body’s ability to process calcium and phosphate into the bones, patients may develop osteoporosis, which is characterized by weak and brittle bones. Osteoporosis puts individuals at a greater risk for fracture. ALERT: Your Tiny Thyroid Causing Hidden Problems: Heart, Obesity, Worse Patients with hyperthyroidism can develop Graves' Disease, which causes visual and eye problems. Without treatment, the disorder worsens. ""People with Graves' ophthalmopathy develop eye problems, including bulging, red or swollen eyes, sensitivity to light, and blurring or double vision. Untreated, severe eye problems can lead to vision loss,"" says the Mayo Clinic. Graves’ disease may even cause problems on the skin, typically around the shins and feet, including swelling and redness. A sudden intensification of hyperthyroidism symptoms is known as thyrotoxic crisis or thyroid storm. It’s typically brought on by some sort of trigger, such as infection, pregnancy, or damage to the thyroid gland. Patients may experience fever, a fast heartbeat, or delirium, according to the Mayo Clinic. Those who experience thyrotoxic crisis should seek medical help immediately. The National Center for Biotechnological Information notes long-term use of some thyroid medications may lead to an increased risk of cancer, according to some studies, though the NCBI said the rise may also be attributed to the thyroid disease itself. SPECIAL: The Real Truth About Iodized Salt the Food Industry Doesn't Want You to Know Related Stories: Thyroid Disorder Symptoms: What Are They? Thyroid Disease: What Is Hyperthyroidism?© 2018 Newsmax. All rights reserved. Email Article | Contact | Print | A APLEASE NOTE: All information presented on Newsmax.com is for informational purposes only. It is not specific medical advice for any individual. All answers to reader questions are provided for informational purposes only. All information presented on our websites should not be construed as medical consultation or instruction. You should take no action solely on the basis of this publication’s contents. Readers are advised to consult a health professional about any issue regarding their health and well-being. While the information found on our websites is believed to be sensible and accurate based on the author’s best judgment, readers who fail to seek counsel from appropriate health professionals assume risk of any potential ill effects. The opinions expressed in Newsmaxhealth.com and Newsmax.com do not necessarily reflect those of Newsmax Media. Please note that this advice is generic and not specific to any individual. You should consult with your doctor before undertaking any medical or nutritional course of action. "


associates degree is how many credits?

How Many Credits Is An Associates Degree? When it comes to the credit scoring to passing the associate degree, it may vary, not much, but in the standard deviation of 10. The minimum credits required for an associate degree is 60, or you can say 90-quarter credits. However, in the majority of the degrees, the requirement is of 70 credits and a few might even require 120 credits. Nevertheless, if a student has earned a decent 60 credits in the Associates degree, he or she is eligible to graduate perfectly.

Misc How Many Credits Is An Associates Degree? By themagreader - September 7, 2015 743Share on Facebook Tweet on Twitter In today’s competitive world, where no one is scared of working hard in his or her lives, it has become so difficult to get a good and high salaried job just on the basis of your high school grades. Moreover, because the high rate of inflations, a high salaried job is the need of the hour without which it is almost impossible to get all the luxuries you want in your life. So, what is the solution? How to get a good and high salaried job? Do you need to study more? The answer is YES. Many of you might have heard about the ‘Associate’s Degree’ and many of you might not. So, if you have not, then definitely some questions will be going through your mind, for example, what is the need of an associate degree, what it takes to get an associate degree, what is the eligibility criteria to get an associate degree, and most importantly, how many credits is an associates degree? We know that these are many questions to be answered but do not worry, we have presented all the answers in the article. So, just sit back, relax and acquaint yourself about the complete knowledge of an Associate degree. Associate Degree- An Overview Before looking into the various important aspects of the associates degree, let us have a brief look and understand what exactly it is. An Associate degree is an academic program that students can take just after their secondary school of education. It typically takes around two years to complete once you enroll yourself into the program. The main goal of an associate degree is to make students aware with some technical, academic and other important skills that are transferable. Therefore, once they are aware of these skills, they can easily get into the jobs of their choices and may go for further studies in the areas, which are their favorite. Once a student earns the associate degree, it means that he or she has completed his or her freshman or sophomore years of the college life. If you are living in the United States or are planning to continue your study there, Associate Degree is one of the most common undergraduate programs, and you may have heard about it. Although, apart from the USA, there are many other countries that offer the associates degree. Some of the countries include Netherlands, Canada, Hong Kong and Austria; however, there are fair chances that the name of the course may be different in different regions. Ares Of Associate Degrees There are various fields where you can have the option to go for an associate degree. An associate degree is generally dis-aggregated by the curriculum and can be of two types- either vocational or non-vocational. Under the vocational programs, there are six categories of associate degree, which include • Paramedical/ health services technology • Engineering/ mechanical technology • Natural Science Technology • Business and Commerce Technology • Data Processing Technology, and • Public Service Technology On the other hand, under the non-vocational degrees there are “General Programs or Arts & Sciences.” The degrees under the non-vocational category are primarily counted by the HEGIS, also known as Higher Education General Information Survey. What After An Associate Degree? This is one of the most important questions, which goes into the minds of students who are willing to go for any course. As the job security is the priority when you go for any course, an associate degree will not disappoint you. There are many fields out there where you can easily get the job of your dreams. Here is a list of areas that are open to you once you complete your associate degree. So, let us have a look into it. • Dental Hygienist • Legal Assistant • Engineering • Air Traffic Controller • Physical Therapist • Operations Technician • Construction Management There is another piece of information that is important for you. It is; you can also go for a bachelor’s degree once you complete your associate degree. Going for a bachelor’s degree will open up the doors for better career opportunities. However, despite the fact that going for a bachelor’s degree is a better option, an associate degree is no less than any other degree and can give you superb career opportunities too. Why Go For An Associate Degree? There are numerous reasons as why students prefer going for the associate degree only. Let us have the look on those reasons. • To get a good job in the young age • The fees for the associate degree is low as compared to other programs and is extremely cost-effective • Wants to go for bachelor’s degree, but could not because of lower grades There are numerous other benefits of going or not for an associate degree. However, it is advisable that before going for it, you do a thorough research about the field you want to go for and check the latest job listings.

However, if you are taking a specialized major, there are possibilities that you may have to fulfill some other important requirements that are the demand of the course. In most of the associates degrees, there will always be some of the core courses or subjects, for example, mathematics, writing, etc. Therefore, it was all about an associate degree. If you are planning to go for an associate degree, it is advisable that you do a thorough research before going for it, especially the courses in trend and the job requirements. SHAREtweet


what is cerebral cavernoma

A cerebral cavernous malformation (also known as cavernous angioma, cavernous hemangioma) is an abnormal group of small blood vessels that may be found in the brain and spinal cord. These lesions can be quiet for many years; however, they can manifest themselves by bleeding. Cerebral cavernous malformations may cause serious neurological symptoms — even death — as a result of severe bleeding or pressure on the brain or nerves.

Cerebral Cavernous Malformation (Cavernous Angioma, Cavernous Hemangioma)â

Cerebral Cavernous Malformation (Cavernous Angioma, Cavernous Hemangioma)Overview Symptoms & Diagnosis Treatment Videos What is a Cerebral Cavernous Malformation?

Common symptoms include: Seizures Headaches Paralysis Cerebral hemorrhage Seizures may be controlled with anti-epileptic drugs. Treatment options vary for cavernous malformations. Surgery may be necessary to remove a cerebral cavernous malformation that is causing symptoms or that suffered multiple bleedings.


what was the dark ages

The Dark Ages were an extraordinarily difficult period in the story of humanity. It is estimated that 100 million people died at the hands of war, poverty, and plague. But during this time new ideas and ideals were born and much of the groundwork was laid for the world we know today.

A Look at the Dark Ages: When Things were Really Medieval The Dark Ages were a period of great upheaval, constant war, horrendous plague, and stagnant cultural growth. But through these difficult centuries new ideas and a new culture was born. And in today’s world we still feel the effects of these changes that were brought about during these Dark Ages. The Dark Ages is a period that is generally accepted as having begun in the year 410 with the fall of Rome and ending in 1095 with the launch of the first Crusades.

The fall of Rome sets a good understanding for what the Dark Ages were all about because for centuries the Roman Empire was a unified force that brought stabilization to most of Europe. It had a vibrant trade and commerce industry that supported a reasonably secure lifestyle for millions of people. When Rome fell, this network of trade and commerce collapsed and the European World was set into chaos. It took seven hundred years of wars, plague, and poverty before the continent came out of it and was moved into the Renaissance. The Black Death- The Middle Ages Poster shown above Medieval Warlords and the struggle to be emperor Before it fell, Rome had been the center of the European world for seven hundred years. The emperor ruled over everything and when this all fell the concept of one man ruling the world still remained. It was this aspiration to rule over everything that perpetuated the darkness of the times. Lords from all over Europe were engaged with each other in battles for land and power. This battling lasted literally centuries and it meant a constant drain of resources and a standstill in cultural growth. Outside Forces make it worse This constant struggling for power within the continent of Europe made it very easy for outside forces to penetrate into the continent and further wreak destruction and drain wealth and resources. From the north Vikings constantly invaded and plundered and from the south Moorish invaders brought war and the word of their prophet. The whole continent was under the constant pressure of three points of attack –from within and from both the north and south. The Plague negates all progress Throughout the first century of the Dark Ages Europe made slow but tangible progress and Emperor Justinian was on the verge of reuniting the continent when the bubonic plague hit and killed tens of millions of people. This destroyed all hope of reunification and kept the continent in chaos for several more centuries. The Force that brought us out of the darkness Christianity was an ideal that rose to power during the dark ages and many warlords of the time embraced it. This had a unifying force on the entire European continent and even though there were many kingdoms they all swore allegiance under the pope. This brought an end to the internal fighting that had been going on for centuries and this unification was solidified with the launching of the Crusades beginning in 1095. This gave all the various warlords and kings a common religious goal and a foe they could join together and focus on. The Crusades, while being for the most part a failure in that they held very little of the land they attempted to conquer, were a significant factor in the rebirth of Europe in that Europe was reunited under a common religion and returning crusaders brought back with them to Europe a wealth of new information in architecture, medicine, philosophy, mathematics and many other areas. This infusion of ideas, paired with the end of constant war within Europe set the stage for the Renaissance.

MOREThe Great Mortality -La moria grandissima began its terrible journey across the European and Asian continents in 1347, leaving unimaginable devastation in its wake. Five years later, twenty-five million people were dead, felled by the scourge that would come to be called the Black Death. The Great Mortality is the extraordinary epic account of the worst natural disaster in European history -- a drama of courage, cowardice, misery, madness, and sacrifice that brilliantly illuminates humankind's darkest days when an old world ended and a new world was born. Weapons and Armor in a Scottish Museum - It is a small museum on the top floor of a building that doubles as a library. But what a gem. They have some amazing weapons and armor including this chilling breastplate. Check it out here The History of Medieval Armor This article takes a look at the medieval period of time between the 5th century and 16th century and how armor developed over this span.